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Description
Coloured by F0x <3

The Lyystranese have a high rate of phenotype variation, leading to many colour variations, as well as other minor physical differences.

Ability wise, the Lyystranese are quite agile, and they are resistant to radiation damage, but only if their diet is laced with heavy metals during adolescence, when they begin to develop the harder chitinous sections on their skin. If damaged enough, the hard chitin will undergo apoptosis and flake off, sometimes coming off in large sections if the damage is extensive. The exposed flesh, given time, will develop and replace the lost chitin, high-z supplements ensure proper replacement of the lost heavy metals that were embedded in the old chitin.

Rarely, the damaged hard chitin will undergo necrosis, rather than apoptosis, and the exposed flesh will not automatically chitinify, increasing the risk of infection. Luckily, initialization of the chitinification process is well understood, and in most cases, the hard chitin can be made to regrow. In a few cases, it will improperly chitinify into the purer, less protective chitin that much of the body is covered by. While this is not deleterious to the health of the organism, the individual will have to adjust to the reduced protective abilities of the softer chitin.

Description
Coloured by F0x <3

The Lyystranese have a high rate of phenotype variation, leading to many colour variations, as well as other minor physical differences.

Ability wise, the Lyystranese are quite agile, and they are resistant to radiation damage, but only if their diet is laced with heavy metals during adolescence, when they begin to develop the harder chitinous sections on their skin. If damaged enough, the hard chitin will undergo apoptosis and flake off, sometimes coming off in large sections if the damage is extensive. The exposed flesh, given time, will develop and replace the lost chitin, high-z supplements ensure proper replacement of the lost heavy metals that were embedded in the old chitin.

Rarely, the damaged hard chitin will undergo necrosis, rather than apoptosis, and the exposed flesh will not automatically chitinify, increasing the risk of infection. Luckily, initialization of the chitinification process is well understood, and in most cases, the hard chitin can be made to regrow. In a few cases, it will improperly chitinify into the purer, less protective chitin that much of the body is covered by. While this is not deleterious to the health of the organism, the individual will have to adjust to the reduced protective abilities of the softer chitin.